Skip Navigation

This Article
Right arrow FREE Full Text (PDF ) Freely available
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (20)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by Ludwig, M.
Right arrow Articles by Diedrich, K.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by Ludwig, M.
Right arrow Articles by Diedrich, K.
Social Bookmarking
Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?


This article has been cited by other articles:


Home page
 J. Clin. Endocrinol. Metab.Home page
A. De Leener, L. Montanelli, J. Van Durme, H. Chae, G. Smits, G. Vassart, and S. Costagliola
Presence and Absence of Follicle-Stimulating Hormone Receptor Mutations Provide Some Insights into Spontaneous Ovarian Hyperstimulation Syndrome Physiopathology
J. Clin. Endocrinol. Metab., February 1, 2006; 91(2): 555 - 562.
[Abstract] [Full Text] [PDF]

Home page
 ReproductionHome page
S. Costagliola, E. Urizar, F. Mendive, and G. Vassart
Specificity and promiscuity of gonadotropin receptors
Reproduction, September 1, 2005; 130(3): 275 - 281.
[Abstract] [Full Text] [PDF]

Home page
 Hum ReprodHome page
A. Delbaere, G. Smits, O. Olatunbosun, R. Pierson, G. Vassart, and S. Costagliola
New insights into the pathophysiology of ovarian hyperstimulation syndrome. What makes the difference between spontaneous and iatrogenic syndrome?
Hum. Reprod., March 1, 2004; 19(3): 486 - 489.
[Abstract] [Full Text] [PDF]

Home page
 NEJMHome page
C. Vasseur, P. Rodien, I. Beau, A. Desroches, C. Gerard, L. de Poncheville, S. Chaplot, F. Savagner, A. Croue, E. Mathieu, et al.
A Chorionic Gonadotropin-Sensitive Mutation in the Follicle-Stimulating Hormone Receptor as a Cause of Familial Gestational Spontaneous Ovarian Hyperstimulation Syndrome
N. Engl. J. Med., August 21, 2003; 349(8): 753 - 759.
[Full Text] [PDF]

Home page
 NEJMHome page
G. Smits, O. Olatunbosun, A. Delbaere, R. Pierson, G. Vassart, and S. Costagliola
Ovarian Hyperstimulation Syndrome Due to a Mutation in the Follicle-Stimulating Hormone Receptor
N. Engl. J. Med., August 21, 2003; 349(8): 760 - 766.
[Full Text] [PDF]

Home page
 Hum ReprodHome page
J.H. Check, J.K. Choe, and A. Nazari
Hyperreactio luteinalis despite the absence of a corpus luteum and suppressed serum follicle stimulating concentrations in a triplet pregnancy: Case report
Hum. Reprod., May 1, 2000; 15(5): 1043 - 1045.
[Abstract] [Full Text] [PDF]

Home page
 Hum ReprodHome page
M. Ludwig, W. Jelkmann, O. Bauer, and K. Diedrich
Prediction of severe ovarian hyperstimulation syndrome by free serum vascular endothelial growth factor concentration on the day of human chorionic gonadotrophin administration
Hum. Reprod., October 1, 1999; 14(10): 2437 - 2441.
[Abstract] [Full Text] [PDF]

Home page
 Hum ReprodHome page
T. Todros, C.M. Carmazzi, S. Bontempo, P. Gaglioti, V. Donvito, and M. Massobrio
Spontaneous ovarian hyperstimulation syndrome and deep vein thrombosis in pregnancy: Case report
Hum. Reprod., September 1, 1999; 14(9): 2245 - 2248.
[Abstract] [Full Text] [PDF]


Disclaimer: Please note that abstracts for content published before 1996 were created through digital scanning and may therefore not exactly replicate the text of the original print issues. All efforts have been made to ensure accuracy, but the Publisher will not be held responsible for any remaining inaccuracies. If you require any further clarification, please contact our Customer Services Department.