Skip Navigation


Hum. Reprod. Advance Access originally published online on March 28, 2007
Human Reproduction 2007 22(6):1503-1505; doi:10.1093/humrep/dem092
This Article
Right arrow Full Text Freely available
Right arrow FREE Full Text (PDF ) Freely available
Right arrow All Versions of this Article:
22/6/1503    most recent
dem092v1
Right arrow Submit a response
Right arrow Alert me when this article is cited
Right arrow Alert me when eLetters are posted
Right arrow Alert me if a correction is posted
Services
Right arrow Email this article to a friend
Right arrow Similar articles in this journal
Right arrow Similar articles in ISI Web of Science
Right arrow Similar articles in PubMed
Right arrow Alert me to new issues of the journal
Right arrow Add to My Personal Archive
Right arrow Download to citation manager
Right arrow Search for citing articles in:
ISI Web of Science (1)
Right arrowRequest Permissions
Google Scholar
Right arrow Articles by vom Saal, F. S.
Right arrow Search for Related Content
PubMed
Right arrow PubMed Citation
Right arrow Articles by vom Saal, F. S.
Social Bookmarking
 Add to CiteULike   Add to Connotea   Add to Del.icio.us  
What's this?

© The Author 2007. Published by Oxford University Press on behalf of the European Society of Human Reproduction and Embryology. All rights reserved. For Permissions, please email: journals.permissions@oxfordjournals.org

Associate editor's comment on the article ‘Semen quality of fertile US males in relation to their mothers’ beef consumption during pregnancy' by Swan et al.

Could hormone residues be involved?

Frederick S. vom Saal1,*

Division of Biological Sciences, University of Missouri, 105 Lefevre Hall, Columbia, MO 65211, USA

1 Tel: +1-573-882-4367; Fax: +1-573-884-5020; E-mail: vomsaalf@missouri.edu

Key words: environmental factors/hormone residues in meat/male infertility/semen quality

The first 150 words of the full text of this article appear below.

In this issue, Swan et al. report finding a relationship between the amount of beef consumed by women during pregnancy and subsequent sperm concentration in their sons in adulthood. There is extensive evidence that maternal nutrition and maternal consumption of specific nutrients, drugs and chemicals present in food during pregnancy and lactation can have consequences for subsequent pathophysiology of offspring. This has been demonstrated experimentally in animals, and the developmental origins of human health and disease (DoHAD) hypothesis also has considerable support from the epidemiological literature (Fowden et al., 2006Go; Gluckman et al., 2007Go).

Only recently have researchers interested in fetal nutrition and metabolic diseases become aware of findings relating developmental exposure to the class of environmental chemicals known as endocrine disruptors to metabolic and reproductive disorders in males. Specifically, Skakkebaek et al. (2001) have identified that a cluster of reproductive abnormalities in males (sperm quality . . . [Full Text of this Article]


Add to CiteULike CiteULike   Add to Connotea Connotea   Add to Del.icio.us Del.icio.us    What's this?